Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ).

Soumyabrata Banerjee; Gary Dunbar

Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ).

Soumyabrata Banerjee, Gary Dunbar

Research output: Contribution to journal › Article › peer-review

Original language	English
Journal	Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357
State	Published - 2021

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@article{e7dfeb126ac04e6db406cd577143d5d3,

title = "Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ).",

author = "Soumyabrata Banerjee and Gary Dunbar",

year = "2021",

language = "English",

journal = "Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357",

publisher = "Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357",

}

Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ). / Banerjee, Soumyabrata; Dunbar, Gary.

In: Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357, 2021.

Research output: Contribution to journal › Article › peer-review

TY - JOUR

T1 - Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ).

AU - Banerjee, Soumyabrata

AU - Dunbar, Gary

PY - 2021

Y1 - 2021

M3 - Article

JO - Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357

JF - Journal of Neurochemistry, March 26, https://doi.org/10.1111/jnc.15357

ER -

Carnosine improves aging-induced cognitive impairment and brain regional neurodegeneration in relation to the neuropathological alterations in the secondary structure of amyloid beta (Aβ).

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