CRISPR-CAS9-mediated non-homologous end joining mechanism for silencing of the mutant huntingtin gene in an in vitro model of Huntington’s disease

N Kolli; Julien Rossignol; Gary Dunbar

CRISPR-CAS9-mediated non-homologous end joining mechanism for silencing of the mutant huntingtin gene in an in vitro model of Huntington’s disease

Research output: Other contribution › peer-review

Original language	English
Publisher	Journal of Cell Transplantation
Number of pages	1
State	Published - Jun 2018

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@misc{c06b7e9d4d1f416db3ba9a1bc7181376,

title = "CRISPR-CAS9-mediated non-homologous end joining mechanism for silencing of the mutant huntingtin gene in an in vitro model of Huntington{\textquoteright}s disease",

author = "N Kolli and Julien Rossignol and Gary Dunbar",

year = "2018",

month = jun,

language = "English",

publisher = "Journal of Cell Transplantation",

type = "Other",

}

CRISPR-CAS9-mediated non-homologous end joining mechanism for silencing of the mutant huntingtin gene in an in vitro model of Huntington’s disease

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