Recurrent Protein-losing Enteropathy and Tricuspid Valve Insufficiency in a Transplanted Heart: A Causal Relationship?

Sanjeev Aggarwal, Ralph E. Delius, Henry L. Walters, Thomas J. L'Ecuyer

Research output: Contribution to journalArticlepeer-review

3 Scopus citations

Abstract

This case report describes a toddler who developed a protein-losing enteropathy (PLE) 4 years after orthotopic heart transplantation (OHT). He was born with a hypoplastic left heart syndrome for which he underwent a successful Norwood procedure, a Hemi-Fontan palliation, and a Fontan palliation at 18 months of age. Fifteen months following the Fontan operation, he developed a PLE and Fontan failure requiring OHT. Four years after OHT, he developed a severe tricuspid regurgitation and a PLE. His PLE improved after tricuspid valve replacement. It is now 2 years since his tricuspid valve replacement and he remains clinically free of ascites and peripheral edema with a normal serum albumin level. His prosthetic tricuspid valve is functioning normally.

Original languageEnglish
Pages (from-to)E10-E13
JournalCongenital Heart Disease
Volume7
Issue number3
DOIs
StatePublished - May 2012

Keywords

  • Heart transplantation
  • Protein-losing enteropathy
  • Tricuspid valve regurgitation

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