Use of CRISPR-CAS9 to reduce levels of mutant huntingtin and mitochondrial dysfunction in an in vitro model of Huntington’s disease

N Kolli, Gary Dunbar

Research output: Contribution to conferencePosterpeer-review

Original languageEnglish
StatePublished - Aug 2018
EventEleventh Biennial Symposium of the Hereditary Disease Foundation - Cambridge, MA
Duration: Aug 1 2018Aug 31 2018

Other

OtherEleventh Biennial Symposium of the Hereditary Disease Foundation
Period08/1/1808/31/18

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